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Taira, Tomi
NENS Committee Member 
Tomi Taira, PhD
Group Leader Neuroscience Center and
Department of Biosciences
University of Helsinki
P.O. Box 65, Viikinkaari 1, Biocenter 3
FIN-00014 Helsinki, Finland
Curriculum VitaeEducation and previous positions- 1995
- Ph. D. in physiology, Univ. of Helsinki
- 1996
- Docent in neurobiology, Univ. of Helsinki
- 1989
- Visiting fellow, NIH/NIAAA,Bethesda, USA
- 1992
- Visiting fellow, Department of Anatomy, Medical School, University of Birmingham
- 1997
- Assistant professor in physiology, University of Helsinki
- 1997
- Visiting fellow, INSERM, Paris
- 2001
- Research fellow, MRC Centre for Synaptic Plasticity, University of Bristol
Other activities- Coordinator, Masters Program in Neuroscience, Univesity of Helsinki
- Invited expert, TEMPUS Masters Program in Neuroscience, University of St. Petersburg, Russia
- Member of Brain Research Society of Finland
- Grant application reviewer, Academy of Finland, Netherlands Organisation for Scientific
Research (NWO, the Dutch research council), Medical Research Council (UK),
Neurosciences and Mental Health Board, CNRS, France, Research Foundation of
the University of Helsinki
| Selected Publications- Segerstrale M, Juuri J, Lanore F, Piepponen P, Lauri SE, Mulle C and
Taira T (2010). High firing-rate of neonatal hippocampal interneurons
is due to attenuation of afterhyperpolarizing potassium currents by
tonically active kainate receptors. J. Neurosci 30:6507-14.
- Sallert M, Rantamaeki T, Vesikansa A, Anthoni H, Harju K,
Yli-Kauhaluoma J, Taira T, Castren E and Lauri SE (2009).
Brain-derived neurotrophic factor controls activity-dependent
maturation of CA1 synapses by downregulating tonic activation of
presynaptic kainate receptors. J. Neurosci. 29: 11294â303.
- Hanse E, Taira T, Lauri SE and Groc L (2009). Glutamate synapse in
developing brain: an integrative perspective beyond the silent state.
Trends Neurosci. 32: 532-537.
- Lauri SE, Vesikansa A, Segerstrale M, Collingridge G, Isaac J and
Taira T (2006). Functional maturation of CA1 synapses involves
activity-dependent loss of tonic kainate receptor-mediated inhibition
of glutamate release. Neuron 50: 415-429.
- Lauri SE, Segerstrale M, Vesikansa A, Maingret F, Mulle C,
Collingridge G, Isaac J and Taira T (2005). Endogenous activation of
kainate receptors inhibits glutamatergic transmission and modulates
network activity in the developing hippocampus. J. Neurosci. 25:
4473-84.
- Lauri SE, Laemsae K, Pavlov I, Riekki R, Johnston B, Molnar E, Rauvala H
and Taira T (2003). Activity-blockade induces formation of functional
synapses in the newborn rat hippocampus. Mol. Cell. Neurosci. 22:
107â117.
- Palva M, Laemsae K, Lauri SE, Rauvala H, Kaila K and Taira T (2000).
Fast network oscillations in the newborn rat hippocampus in vitro. J.
Neurosci. 20: 1170-78.
- Laemsae K, Palva M, Ruusuvuori E, Kaila K and Taira T (2000). Synaptic
GABAA activation inhibits AMPA/kainate receptor-mediated bursting in
the newborn (P0-P2) rat hippocampus. J. Neurophysiol. 83: 359-366.
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